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Air Max Pas Cher As documented by over 2200 publications in the PubMed database and summarized in numerous recent reviews,5, 8, 9, 10, 11, 12, 13 GC induce massive apoptosis in certain cells of the lymphoid lineage, particularly immature thymocytes and ALL cells, and the latter has been exploited in the therapy of lymphoid malignancies.4 GC have further been reported to induce cell death (alone or in combination with other death inducers) in some nonlymphoid tissues and cells such as bone,14 hippocampus,15 eosinophils,16 fibroblasts17 and certain cancer cells.18 Interestingly, GC support survival in erythroblasts,19 neutrophils20 and several nonhematologic tissues such as mammary gland, ovary, liver and fibroblasts (reviewed in Amsterdam and Sasson21).
Nike Air Foamposite Such prosurvival effects may become clinically relevant when they interfere with the effect of chemotherapeutics.22 Depending upon the circumstances, GC both triggered cell death and supported survival in some cells,23 further documenting the pro and antiapoptotic potential of this hormone.General mode of action of GCAlthough receptor independent effects may occur at very high concentrations (presumably through membrane perturbation24), most,oakley outlet if not all, effects of GC at physiologic or therapeutic levels are mediated by the GC receptor (GR, Figure 1).